STD eats flesh “down there” – fact or fiction?
| FOX NEWS – A rare sexually transmitted disease that causes flesh eating ulcers on patients’ genitalia has popped up in England.
An unnamed female patient was diagnosed with donovanosis within the last 12 months.
Oh, it’s real alright …
Donovanosis (also known as Granuloma inguinale) is spread through sexual intercourse with an infected patient, or by coming into contact with a patient’s infected ulcer.
The virus is typically seen in India, New Guinea, parts of the Caribbean, central Australia and southern Africa.
According to the CDC, the disease causes progressive ulcerative lesions on the genitals or perineum, which are prone to heavy bleeding.
Patients are at risk of infections not only on the genitals but in the pelvic regions, or in intra-abdominal organs, bones or mouth. The lesions may also develop secondary bacterial infections.
While antibiotic treatment may stop the progression of lesions, patients are at risk of relapse for 6-18 months post-treatment. Read more at Fox News. Coverage continues below …
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From Wikipedia, the free encyclopedia
Granuloma inguinale (also known as donovanosis) is a bacterial disease caused by Klebsiella granulomatis (formerly known as Calymmatobacterium granulomatis) characterized by genital ulcers.
It is endemic in many less developed regions.
It is also known as donovanosis, granuloma genitoinguinale, granuloma inguinale tropicum, granuloma venereum, granuloma venereum genitoinguinale, lupoid form of groin ulceration, serpiginous ulceration of the groin, ulcerating granuloma of the pudendum, and ulcerating sclerosing granuloma.
The disease often goes untreated because of the scarcity of medical treatment in the countries in which it is found.
In addition, the painless genital ulcers can be mistaken for syphilis.
The ulcers ultimately progress to destruction of internal and external tissue, with extensive leakage of mucus and blood from the highly vascular lesions. The destructive nature of donovanosis also increases the risk of superinfection by other pathogenic microbes.
Small, painless nodules appear after about 10–40 days of the contact with the bacteria. Later, the nodules burst, creating open, fleshy, oozing lesions. The infection spreads, mutilating the infected tissue.
The infection will continue to destroy the tissue until treated. The lesions occur at the region of contact typically found on the shaft of the penis, the labia, or the perineum.
Rarely, the vaginal wall or cervix is the site of the lesion. At least one case in India led to partial autoamputation of the penis.
The patient tested positive for HIV-2 and had been infected for six years.
The microorganism spreads from one host to another through contact with the open sores.
The diagnosis is based on the patient’s sexual history and on physical examination revealing a painless, “beefy-red ulcer” with a characteristic rolled edge of granulation tissue. In contrast to syphilitic ulcers, inguinal lymphadenopathy is generally mild or absent.
Tissue biopsy and Wright-Giemsa stain are used to aid in the diagnosis. The presence of Donovan bodies in the tissue sample confirms donovanosis. Donovan bodies are rod-shaped, oval organisms that can be seen in the cytoplasm of mononuclear phagocytes or histiocytes in tissue samples from patients with granuloma inguinale.
They appear deep purple when stained with Wright’s stain.
These intracellular inclusions are the encapsulated Gram-negative rods of the causative organisms. They were discovered by Charles Donovan. Read more at Wikipedia.